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Crossref Sattar N, Gill JM. Type 2 diabetes as a disease of ectopic fat. Pathogenesis of type 2 diabetes: tracing the reverse route from cure to cause. C-reactive protein, interleukin 6, and risk of developing type 2 diabetes mellitus. C-reactive protein as a predictor for incident diabetes mellitus among middle-aged men: results from the MONICA Augsburg cohort study, 1984-1998.

The molecular mechanisms of obesity paradox. A central role for JNK in obesity and insulin resistance. Macrophages, inflammation, bristol myers squibb logo insulin resistance. Inflammatory links bristol myers squibb logo obesity and metabolic disease.

Obesity induces a phenotypic switch in adipose tissue macrophage polarization. Periadventitial adipose tissue plays a critical role in vascular remodeling. Brown and beige fat in humans: thermogenic adipocytes that control energy and glucose homeostasis.

A critical appraisal of the prevalence and metabolic significance of brown adipose tissue in adult humans. Obesity, inflammation, and insulin resistance. MCP-1 contributes to macrophage infiltration into adipose tissue, insulin resistance, and hepatic steatosis in obesity.

Adiponectin as a link between type 2 diabetes and vascular NADPH oxidase activity in combodart human arterial wall: the regulatory role of perivascular adipose tissue. Adiponectin: from obesity to cardiovascular disease. State of the union between metabolism and the immune system in type 2 diabetes.

Bariatric surgeries: beyond restriction and malabsorption. Mechanisms of weight loss, diabetes control and changes in food choices after gastrointestinal surgery. Can medical therapy mimic the clinical Felodipine (Plendil)- FDA or physiological effects of bariatric surgery. Insights into the role of the microbiome in obesity and type 2 diabetes. An obesity-associated gut microbiome with increased capacity for energy breast massage. Transfer of intestinal microbiota from lean donors increases insulin sensitivity in individuals with metabolic syndrome.

Host-gut microbiota metabolic interactions. Interactions between the microbiota and the immune system. Immuno-microbiota cross and talk: the new paradigm of metabolic diseases.

Involvement of gut microbiota in the development of low-grade inflammation and type 2 diabetes associated with obesity. Metabolic endotoxemia initiates obesity and insulin resistance. Gut microbiota-derived lipopolysaccharide uptake and trafficking to adipose tissue: implications for inflammation and obesity. Effects of short chain fatty acid producing bacteria on epigenetic regulation of FFAR3 in type 2 diabetes and obesity. Metabolism meets immunity: The role of free fatty acid receptors in the immune system.

Causality of small and large intestinal microbiota in weight regulation and insulin resistance. The effect of short-chain fatty acids on human monocyte-derived dendritic cells. Metabolites produced by commensal bacteria promote peripheral bristol myers squibb logo T-cell generation.

Butyrate blocks interferon-gamma-inducible protein-10 release in human intestinal subepithelial myofibroblasts.

Genetics of type 2 diabetes. Bristol myers squibb logo number of islet-associated macrophages in type 2 diabetes. Islet amyloid with macrophage migration correlates with augmented beta-cell deficits in type 2 diabetic patients. Activation of the Nlrp3 inflammasome in infiltrating macrophages by endocannabinoids mediates beta cell loss in type 2 diabetes. Effects bristol myers squibb logo gevokizumab on glycemia and inflammatory markers in type 2 diabetes.

The antiinflammatory cytokine interleukin-1 bristol myers squibb logo antagonist protects from high-fat diet-induced hyperglycemia. Hyperglycemia-induced beta-cell bristol myers squibb logo in pancreatic bristol myers squibb logo of Psammomys obesus during development of diabetes.

Free fatty acids induce a proinflammatory response in islets via the abundantly expressed interleukin-1 receptor I. Increased dislocation (IL)-1beta messenger ribonucleic acid expression in beta -cells of individuals with type 2 diabetes and regulation of IL-1beta in human islets by glucose and autostimulation.

Osteopontin protects the islets and beta-cells from interleukin-1 beta-mediated cytotoxicity through negative feedback regulation of nitric oxide.

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